The HD protein causes a depletion in cellular energy but not through direct effects on the mitochondria, the cell's energy factory.

Exciting research suggests that restoring the expression of the genes that the HD protein suppresses could be a major treatment.

The molecular zip code research suggests that a kinase inhibitor could be a major treatment for Huntington

Researchers generated a mouse which progressively lost Dopamine 1 receptor cells and got Huntington's Disease like symptoms.

Researchers have discovered that excess copper plays a role in Huntington's Disease pathology.

New findings based on new technology show that the HD protein is being misdirected within the cell. Small molecules are being developed which might place a 'molecular zip code' on the problem.

Problems with NCAMs may explain cognitive and olfactory dysfunction in HD.

Stem cells are still present in the middle aged brain; they just aren't dividing.

A company currently doing stem cell research in animal models of HD, has filed an application with the FDA for permission to begin clinical trials of stem cell treatments for stroke victims.